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Dr Trevor Marshall
Foundation Staff
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Posted: Mon Jan 8th, 2007 04:02 |
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Norman,
Academics interact a little bit differently from the average "man-in-the-street." The welcome I got at University of Melbourne was warm, and my discussion with the attendees at the drinks-gathering after the presentation was exceptionally stimulating.
I took the context of the 'long road to hoe' (in the the closing remarks) as being with reference to Barry Marshall's Nobel Prize, and few would argue that we have some years still to go before acceptance of the MP to the level of Nobel.
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tickbite
Guests visiting Phase 1/2/3
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Posted: Mon Jan 8th, 2007 18:20 |
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Plasmids..........
Transposons..........
Also, I know this has nothing to do with this thread but this is interesting news:
Antibody Therapy Prevents Type 1 Diabetes In Mice
~Grego
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"Lyme","CFS", Meningitis
Phase3 8-2-07, MP on hold 11/2007
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norman
Member in Phase 3
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Posted: Wed Jan 10th, 2007 02:01 |
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| Thanks for the info and the article. I guess I am not sure what to make of the article. The article seems to indicate a genetic problem, correct?
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Sarc| Ph3 approx 6/1,Acne/ increase skin lesions, anxiety/depression /High IgG and Iga for C. Pneumoniae,last d= 10
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norman
Member in Phase 3
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Posted: Wed Jan 10th, 2007 02:08 |
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Ah, thanks for the reply Trevor. I guess I seemed to take it differently, as if he was saying " novel idea, but not for us" but I guess I interpreted those marks differently.
I wish you the best of luck (obviously- lol). I have to tell you, it seems to me that I am seeing, what seems to me, to be more and more people with autoimmune disorders! I have told quite a few people about your site, and hopefully my aunt will be starting this soon ( she's getting close to getting approval from doc I hope !)
Hopefully there will be another conference soon so I can meet more of the wonderful people I am talking to on here.
Take care!
Norman
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Sarc| Ph3 approx 6/1,Acne/ increase skin lesions, anxiety/depression /High IgG and Iga for C. Pneumoniae,last d= 10
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tickbite
Guests visiting Phase 1/2/3
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Posted: Wed Jan 10th, 2007 19:57 |
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Hey Norman,
The only reason why I would think that the antibodies worked "when the researchers transferred a certain subset of T-cells from anti-CD137-treated mice that expressed two other receptors known as CD4 and CD25 to other immune-deficient NOD mice, it prevented the onset of diabetes in the recipient mice" (S.D. http://www.sciencedaily.com/releases/2007/01/070108121653.htm)
is because "An antibody or immunoglobulin is a large Y-shaped protein used by the immune system to identify and neutralize foreign objects like bacteria and viruses. Each antibody recognizes a specific antigen unique to its target." (wiki http://en.wikipedia.org/wiki/Antibody)
Genetic predisposition? or infectious genetics? or CWD forms embedded within the phagocytes? IMO, normal base rate mutation is a blip compared to infectious mutation occuring in mankind these days.
I'm not for sure though, i'm still researching genetics and i'm an amatuer. Mutagens have been catching my eye recently.
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"Lyme","CFS", Meningitis
Phase3 8-2-07, MP on hold 11/2007
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