The Marshall Protocol Study Site > PROF. MARSHALL'S PERSPECTIVE > Prof. Marshall's Perspective > Bacterial Iron Acquisition Systems
|
|
The Marshall Protocol Study Site > PROF. MARSHALL'S PERSPECTIVE > Prof. Marshall's Perspective > Bacterial Iron Acquisition Systems |
|
| Moderated by: Dr Trevor Marshall | ||
| Author | Post | ||||||||||
|---|---|---|---|---|---|---|---|---|---|---|---|
|
P.Bear R.N. Inactive Staff
|
I found this interesting, and it may help explain why so often serum levels of iron will test low in chronic infectious states; and why replacement may not usually be a good idea. P.B. http://www.biosci.utexas.edu/mgm/People/Faculty/profiles/payne.htm |
||||||||||
|
Dr Trevor Marshall Research Team 
|
Thanks PB, thats an excellent explanation of why Th1 folk should not be unduly worrying about their low iron. And it allows perfect meshing between obesity and low iron levels, something the authors of this study were unable to do http://www.reuters.com/article/healthNews/idUSN2934050920070904 ..Trevor.. |
||||||||||
|
Freddie Ash Member in Phase 3 
|
HI PBEAR This is Fred in WV. I want to share a story about low iron. Back in July 2005 I was in a restroom with a nosebleed and a man came in and started talking to me about the nosebleed. I told him that I had a disease called sarcoidosis that caused it. He then told me his wife was in a local hospital because she had Lupus. He said her iron got so low they put her into the hospital and started giveing her something for it and it made her worse. I shook my finger and said,"YOU DOG GONE RIGHT IT WILL. WITH THESE DISEASES WE HAVE LOW IRON AND IF YOU TAKE SOMETHING FOR IT, IT CAN MAKE YOU WORSE." I told him the best thing was to just keep a close eye on it, but she should get on the Marshall Protocol and I told him how to find it on the internet. Remember, we are all in this together and I am pulling for us. Your friend in sarcoidosis Freddie |
||||||||||
|
Lee Member in Phase 3 
|
I too have the low iron values ....and have just started using my iron skillets more and more. Is this wrong? Should I toss these skillets? Thanks! Lee |
||||||||||
|
P.Bear R.N. Inactive Staff
|
Lee, I might not toss the lovely cast iron, but would avoid cooking acidic foods that would absorb much more iron. We need some iron and I don't think we need to be total fanatics about avoiding foods that have iron. I think we just don't usually want to take any supplements unless it is absolutely determined to be medically necessary; and slightly low levels should not be the indication to replace. I found a link that shows that iron cookware does indeed increase iron in foods, and that acidic foods tend to suck up the most iron here: http://whatscookingamerica.net/Information/IronCastIron.htm Best, P.B. |
||||||||||
|
Lee Member in Phase 3
|
Thanks PB! Good to know as some of them are family heirlooms .... Lee |
||||||||||
|
Grace Member in Phase 3
|
DH's iron was real low since he got sick for years.The medico people all said "your a male, your iron should not be this low with out losing blood".He had many tests over the years to try to find a source of blood loss.....there was none of course.He was place on iron tabs, which didn't do a lot for his iron levels.He stopped these when started MP.Then his iron went through the roof.Doc 'freaked',I just said "Isn't that interesting ",and reassured him it will come back down...and it has.Grace |
||||||||||
|
Joyful Board Staff 
|
Late Saturday morning regularly finds my husband in the back room of the old fly shop across town. The owners are in their eighties and enjoy the small gathering of old friends that show up pretty often to engage in small talk on many subjects including fly fishing. The wife has low iron. The doctor's solution has been to give her a blood transfusion every month or so. This really perks her up for a while. The joke last week is that since the blood was from all types of donors and most likely many young donors, she is increasing her diversity and reversing her age! Lots of Th1 "clues" with many of the folks in that group. |
||||||||||
|
Dr Trevor Marshall Research Team
|
Joyful, as far as I can see, Th1 pathogens start to dictate the 'health' of just about everyone as they age. If you draw a graph of 25-D levels vs age, they drop steadily after age 40. Something is happening, even during 'healthy aging', that we really ought to understand a little more There is a branch of medicine which is starting to look at Immunity and Ageing. Here is a short letter I recently wrote to the editor of one of the journals: http://www.immunityageing.com/content/3/1/12/comments |
||||||||||
|
Joyful Board Staff
|
Perhaps someone will stand up and take notice in this arena! Your letter spells it out well enough Dr. Marshall, but people just don't have "grid" to taking the information in.  (You could start your own fiction series to popularize the concepts, I suppose.) Back to the topic of pathogens and iron... A few years before the Crohn's Disease symptoms became obvious for one of the male members of my family, he was tested for an abnormal gene for Hemochromatosis. I guess it is thought that some people with this genetic abnormality will end up with serious organ damage from iron overload. Their words were, "...in you the gene abnormality is located in a section that only occasionally causes trouble. ... The technical description of all this is that you are homozygous for Hemochromatosis at the 187-nucleotide locus of the gene." They went on to recommend testing every 5 years for iron overload for the rest of his life. They also recommended avoiding high doses of vitamin C, multivitamins containing iron, excessive alcohol consumption, and they suggested getting immunizations against Hepatitis A & B! I am curious about the reason behind their immunization recommendations. I would be interested to see if these types of "genetic defects" would retest the same following a long term treatment with the MP. From the reading here on the study site, I would suspect that the genes have been "broken" by the infectious organisms and have the potential to be restored if the infectious agents were eliminated. Is that possible? |
||||||||||
|
scooker48 Member in Phase 3 
|
This is very interesting. If one cooks with stainless steel cookware, made of chromium, nickel and iron, it has an oxide layer of chromium which protects the food. Or so the metallurgists explained to me in the hallway just now. However, if one is cooking an acidic food, such as tomatoes, it would dissolve the oxide layer faster. I think the important thing is not take supplements, but the cookware issue is "food for thought"...or "food for the bacteria"? LOL Sherry |
||||||||||
|
Martin78 Member in Phase 1 
|
Hi Since I started feeling (really) sick in May I have tested Iron, Trensferrin and something called "transferrinsaturation" three times. My Iron and Tranferrin is normal, but my "transferrinsaturation" is low at 16% (ref range 15%-57%). I remember to have read something in the "bloodwork" section about bacteria needing iron to grow. I wonder if it is this that is shown in the low transferrin saturation? BR Martin |
||||||||||
|
Foundation Staff . 
|
See My doctor says I'm anemic. What should I do? |
||||||||||
|
Martin78 Member in Phase 1
|
Thank you Meg I will print it out and ask my doctor to educate me on this. I read the thread, but I am not sure if my levels are according to what you expect in chronic innflamations. I didnt know that my low ferritinsaturation was making me anemic. My iron is lower end at 13,4 (9,0-34,0) Transferrin is normal 3,4, and my Hb is alway normal from 14,5-15,7. Its seems like you are working alot nowadays so I have restrained myself from asking to many questions. ( I ask them in the new site) but the limited medical ones I dare myself to aks here at mp.com. Hopefully I will be able to reopen my "Martin on Benicar" thread soon. You are doing a great job! |
||||||||||
|
Cocoa Member in Phase 2/3
|
Hi All, I've found this thread really interesting because eversince I became unwell I've had consistently low ferritin in every blood test I've had. Prior to becoming unwell my ferritin was always approximately 100 or more. Since being unwell, it has varied from 25 to 60... and this was with regular supplementation! When I read the information on the site about the bugs utilising the iron, it made a lot of sense why my iron has been low since being unwell. BUT, what is really fascinating is that since starting MP I have stopped ALL iron supplements (as is necessary) and on my first blood test two weeks after starting MP my ferritin when up to 104!!! And the next test showed it to be 66. Wow! And please bear in mind that I stopped supplementing iron which I was doing pretty regularly. That makes me smile . Cocoa |
||||||||||
|
Claudia Member in Phase 3 
|
When I was diagnosed with hypothyroidism (from Hashimoto's thyroiditis) I had iron-deficiency anaemia. Doctor ordered me to take iron supplements for a month before he even gave me any thyroxine. BIG mistake. (Not his fault, but had we only known about the MP...) My iron levels came up, yes, I guess because I was ingesting enough to feed myself and all those iron-hungry bacteria! The awful part was I felt more fatigued than ever before and my arthritic joints hurt more than ever before. I was depressed, in pain and had to give up my work as a massage therapist after 25 years. Once I started thyroxine my life turned around, as I got most of my energy back and my thinning hair started growing back - but my arthritis was as bad as ever. Fast forward 1 year - I found the MP and began phase one. Within days of starting mino I began to experienc a metallic taste in my mouth. Could that metallic taste be ... metal? Can't prove it, but my ferritin levels shot through the roof! You can read my phase 1 posts on my thread: http://www.marshallprotocol.com/forum20/5902-1.html I reckon all those iron-sequestering CWD monsters were dying and giving up all the iron back into my bloodstream. After a couple of months, my ferritin levels came down to normal again and the metallic taste went away at the same time. So there you have it.  Claudia |
||||||||||
|
Cocoa Member in Phase 2/3
|
Hi Claudia, That is a really interesting idea... my first blood test a few weeks into the MP saw my ferritin levels shoot right up to just on the upper limit of normal (after having been on the other end of the spectrum for years) and then subsequent tests a few months later show the ferritin being half as much as that initial test but still well within normal limits. I didn't get a metallic taste... but your idea may explain why the levels shoot up and then go back to normal! My cheers, Cocoa |
||||||||||
|
Foundation Staff .
|
When I was diagnosed with hypothyroidism (from Hashimoto's thyroiditis) I had iron-deficiency anaemia. Doctor ordered me to take iron supplements for a month before he even gave me any thyroxine. BIG mistake. (Not his fault, but had we only known about the MP...) Iron depletion (low serum ferritin) does not prove iron deficiency anemia. Anemia is common in chronic disease and iron deficiency anemia should be verified before an iron supplement is ordered. See My doctor says I'm anemic. What should I do?
Periodic increases in ferritin which correlate with MP meds suggests liver inflammation. |
||||||||||
|
Kas Member in Phase 2 
|
Amazingly, one of the things my doc has NOT worried about, is my low iron levels. She learned in med school that folk with autoimmune diseases often have it and that we should not be supplemented. She says she would only worry if my haemoglobin levels went too low. My niece in Australia was 24 when she got colon cancer. She also has celiac disease. It is now a year after her surgery and chemo ,and her iron levels are so low, she goes into the hospital for transfusions, as the pills do nothing to help. I wonder if they are doing the right thing for her? Even with my low iron levels, I have never suffered the fatigue others seem to. I am one weird specimen, that's for sure!!! |
||||||||||
|
Foundation Staff .
|
Low Hgb and Hct are also not a cause for concern in someone with chronic disease. Fatigue is not caused by low stores of iron. |
||||||||||
|
Kas Member in Phase 2
|
Interesting. I wonder why so many doctors claim that being low in iron makes one fatigued and low in energy? Look at how many folk are swallowing those darn iron pills which give them gastric problems because medical people think it will make them feel much better. |
||||||||||
|
Ruth Goold Health Professional
|
Meg, Is it possible that a person with (supposedly dangerously) high ferritin levels also suffers from Th1 inflammation? Ruth |
||||||||||
|
Foundation Staff .
|
Serum ferritin may be high in chronic inflammation, especially if the liver is involved. This article lists the other tests done when a differential diagnosis is needed. Anemia of chronic disease equals: -ferritin normal or high -iron low -normal to low soluble transferrin receptor (sTfR) -normal to low total iron-binding capacity (TIBC) A high ferritin level suggests further investigation should be done with the D-metabolites tests. |
||||||||||
|
Ruth Goold Health Professional
|
Thanks very much Meg. I'm working on yet another relative with likely Th1 problems. He's had the D tests done: certainly indicative of possible Th1 inflammantion with 25-D ~20 ng/ml and 1,25-D ~42 pg/ml (he's on no meds and does no supplementation of D or anything else). Of course, he's been told that both tests are 'perfectly normal'. He donates blood to bring his ferritin levels down but they have been very high for several years and I am concerned about him developing liver cancer. He is currently waiting to see me improve on the MP (unfortunately, I am a spectacular case of one who seems to change very slowly). Before the MP, I was tired. Still tired. Can't honestly tell anyone that I think that I have changed (although I certainly hope that I have ).As always, I'll print out the info and pass it on. Thanks for your tireless efforts, Ruth |
||||||||||
|
Claudia Member in Phase 3
|
Periodic increases in ferritin which correlate with MP meds suggests liver inflammation. aha! I knew my liver must be involved. But as for my "anaemia" - well, "it just gets curiouser and curiouser", as Alice said. re: Iron depletion (low serum ferritin) does not prove iron deficiency anemia. Anemia is common in chronic disease and iron deficiency anemia should be verified before an iron supplement is ordered. See My doctor says I'm anemic. What should I do? in that thread suggested above, Belinda writes: One of the potential 'benefits' of the anemia of chronic disease is that bacteria are being starved of the iron essential for their proliferation. Once CWD bacteria are killed off, iron stores will be available for your own body once again. so, I think I am getting that: 1-anaemia, in chronic disease is a defensive move on the part of my body - to starve the bacteria. 2- that explains why iron supplementation (as I certainly experienced) will fail and even exacerbate the illness. 3- there is agreement with my statement that the MP frees up iron; but it is not clear where it is coming from; if that is what is showing up as ferritin levels rising or what. Can someone clarify why this is so, and how the liver is involved? Is the liver storing the iron to keep it from the bacteria? I still wonder what happens to the iron/ferritin contained in the red blood cells when they are sacrificed during cell-death. Does it float around in the blood? End up in the liver? or elsewhere? hmmm... Regarding my anaemia, I can recall my doctor telling me that at the time I first presented with hypothyroidism, I had Megaloblastic anaemia. I have no idea if iron supplementation is the correct treatment for that - perhaps not. (I think my body functions were all shutting down due to lack of thyroid hormone at the time.) My ferritin at the time was 34 and my iron was 15. Iron supplementation seemed to temporarily boost these, but a month later both had dropped again - the iron dipping to its lowest, at 9. hmm.... rebounding due to flourishing CWD bacteria? My Haemoglobin, Haematocrit, red cell count and white cell counts were all in normal range until I began the MP, more than a year later. In the course of the MP they all have dropped into "Low" levels, which I suppose indicates apoptosis, or programmed cell death of infected blood cells - would that be right? So that is my latest kind of anaemia. Does it have a name? Shall we dub it "Temporary Anaemia of MP Therapy"? |
||||||||||
|
ndodd Health Professional
|
Megaloblastic anaemia means reduction in numbers of red cells but an overall increase in their size, ie they are abnormally large. Can be associated with B12 deficiency, alcoholism, or for no known reason. Iron deficiency aneamia has small cells with reduced haemoglobin content (low MCV and low MCHC), so these two aneamias are quite different. An 'anaemia of chronic disease' does seem to be common to phase 3 patients. Personally I dont think its cell death. Red cells are replaced every 3 months, and white cells every 4 months I think. So your body has slowed down the replacement of them, IMO because its caught up in a much bigger fight and is 'tired'. Therefore the importance of ramping cautiously. |
||||||||||
|
P.Bear R.N. Inactive Staff
|
I also think that the Benicar may be boosting renal blood flow and renal tissue oxygenation enough that the production of erythropoietin is down regulated and there is some consequent reduction in the stimulus to produce red cells (that is not too profound). I do think that herx/immunopathology can at times either wipe out red cells or effect their production in certain people due to endotoxin effects. http://ajplegacy.physiology.org/cgi/content/abstract/231/1/73 best, P.B. |
||||||||||
|
Caitiegirl Member in Phase 2
|
I feel like my life has just traveled in a circle. Twenty something years ago I was in Grad school studying Vibrio cholera and Vibrio vulnificus. While almost everyone has heard of Cholera, V. vulnificus is a particuarly nasty food borne illness. It can also be contracted by swimming in infected waters with an open wound. The research in our lab at that time was showing that the bacteria's ability to obtain iron from the host was one of the major, if not the major determining factor in mortality. I remember the case of an alcoholic with cirrhosis of the liver who was standing on an ant hill. He ran into the ocean to wash the ants off his legs. The bacterium entered through the ant bites and he was dead in days. Only people with high serum ferritin levels responded this way to this infection. I am sure the research into the mechanism of siderophores in this species of bacterium has continued and progressed since I was there. Sadly I have not kept up with the science and this was not my area of expertise. I was supposed to develope a probe which would find "viable but non-recoverable" V. vunificus in seafood and the environment. Sounds a little like CWD? It was later found that in the VBNR state bacteria had significant changes in their cell wall structure and became coccoid (vibrio is rod shaped normally). To get back on subject, it is very easy for me to believe that low iron levels is the body's adaptive response to prevent the bacterial infection from thriving. I think this is just another example of doctor's having test results but viewing them in a two dimensional, simplistic fashion. Maybe it's not the cause of the fatigue but the result of the disease process causing the fatigue. JMHO, Mindy |
||||||||||
|
schesche inactive member
|
Belgian Rickettsial researcher and pioneer for occult infections Dr JAdin allready cited low iron as a consequence of chronic rickettsial infection--in 1955 along with paul giroud ,paul Legag |
||||||||||
|
Cocoa Member in Phase 2/3
|
I was watching an Australian science program this week. It focussed on the resurgence in TB due to the bugs developing abx resistance. The program was fascinating stating that TB eschews the immune system by hiding in the macropages and, more relevant to this topic, that a new drugs is being developed to combat TB by preventing the bugs from getting access to the host's iron which is fundamental to their survival. Very interesting! You can watch it at: http://www.abc.net.au/catalyst/stories/2395725.htm Does anyone think that this could be applied to others suffering from CWD infection... by the way I wonder if TB gets into the macrophages by converting to the L-form? Best, Cocoa .Last edited on Sat Oct 25th, 2008 16:16 by Cocoa |
||||||||||
|
Dr Trevor Marshall Research Team
|
Mycobacteria use a different mechanism to evade the immune system than the metagenomic microbiota (biofilm) which causes Th1 disease. The resurgence of TB is actually due to a surge in Th1 disease, which knocks out the innate immune system. The 'experts' will figure this out, eventually. I did have a sentence or two about this in my Bioessay: http://TrevorMarshall.com/BioEssays-Feb08-Marshall-Preprint.pdf |
||||||||||
|
Rico Moderator
|
So, TB mycobacteria are co-infections? Boy, it's been one deception after another...it's incredible to see how medicine has been side-tracked all these years ... and all of the suffering and gazillions of dollars spent on studies, all the while the root cause was just continuing to strive and making things worse ... sigh ... food for thought. |
||||||||||
|
Dr Trevor Marshall Research Team
|
The microbiota have been around since Neolithic times, so, as a civilization we have come to expect nothing except what they deal us. Just today there was news that the same mutation caused several, apparently unrelated cancers, it has been tough to put 2 and 2 together until we started to really understand the metagenome. http://www.reuters.com/article/healthNews/idUSTRE49N83D20081024 |
||||||||||
|
NickBowler Member in Phase 3 
|
Here are a couple of articles I posted back in Feb in another thread. Hepcidin is an AMP which is regulated by the VDR. When the VDR is blocked, hepcidin levels drop and serum iron rises because hepcidin is a major controller of the iron withholding system in the body. The TH1 bacteria can then thrive. Just another piece in the puzzle! I had high transferrin saturationand serum iron for quite a while before I found the MP, but a few months after starting I had developed megaloblastic anemia. http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=308925 http://bloodjournal.hematologylibrary.org/cgi/content/full/102/3/783 |
||||||||||
|
geirf Health Professional
|
And we get them as a birth present , at least some of them ? The infection route is rather difficult for most of them I guess if they don`t get help from Ticks or mosquitos ?? Or blood transfusions and vaccinations ?? what about eating raw meat or fish ?? My main patient population are autists.. Have there been any look into how many of the autists having Th1 disease - or intracellular metagenome as a cause ? TO my knowledge that have not been looked at, and causes are mainly stated to be viral, and or toxicological combined with genetic susceptibility . |
||||||||||
|
Dr Trevor Marshall Research Team
|
Geirf, Yes, all of the above sources. Please take the time to look at the new videos of my two presentations in China over the Holidays. This exact question of where the bugs come from was canvassed in my presentations and in the question and answer sessions (the seminar at West China Hospital will be put online later today, my Keynote at the Gene 2008 conference is already online).We have several 'autistic' ADHD and ASD children. We have not been recruiting for a number of reasons, but I can tell you that they respond with Immunopathology just as the adults with the rheumatic diseases do, and the kids on the MP have started on their paths to recovery, by, at least, getting back to school - and their classmates Although Matt was primarily beset by 'tics' and not true ASD, you can read his recovery story here: http://bacteriality.com/2007/10/28/interview6/ A Very Merry Christmas and many Happy New Years to you and yours |
||||||||||
|
Lottis Health Professional 
|
Dr Inger Mattsby-Baltzer at the clinical bacteriology department of Sahlgrenska, is doing some interesting research from Goteborg, Sweden. Peptides taken from a large molecule in human maternal milk, Lactoferrin, has been indicating to inhibit the fibrosis that often appears in scarring and especially in the abdomen connected to IBD (inflammatory bowl diseases). http://www.google.com/patents?hl=en&lr=&vid=USPAT7253143&id=-86AAAAAEBAJ&oi=fnd&dq=Inger+Mattsby-Baltzer Testing on patients, with these peptides will begin at the end of 2009. http://www.nyteknik.se/nyheter/bioteknik_lakemedel/lakemedel/article497055.ece I dug a little bit futher into lactoferrin, and found this paper from 2007, that studies cases of iron depletion in prolonged neutropenia and cases of chronic granulomatous disease (CGD). Human Polymorphonuclear Leukocytes Inhibit Aspergillus fumigatus Conidial Growth by Lactoferrin-Mediated Iron Depletion http://www.jimmunol.org/cgi/reprint/178/10/6367 Very interesting!
|
||||||||||
|
Lottis Health Professional
|
Professor Kjell Olmarkers findings was the driving force, to find the peptides for preventing the fibrosis. http://journals.lww.com/spinejournal/pages/articleviewer.aspx?year=2004&issue=11150&article=00002&type=abstract |
||||||||||
|
Lottis Health Professional
|
Lottis wrote: Dr Inger Mattsby-Baltzer Both of the discussions at those sites, refer mostly to fungus. I wonder if maybe fungus require more iron than many other pathogens. That could mean that if somebody has more fungus pathogens, the less levels of iron will be detected in the persons blood. More fungus = Less iron? Pregnant women often get infected with fungus, since the pH in the mucus often go more basic during pregnancy. Many other factors has made fungus infection quite common among young girls, as well. And also iron deficiency. To heal the imbalance of fungus and other pathogens, breast feeding will supply the child with the weapons to balance and normalize the little baby. We know this, but maybe, now we know even more... /Lottis Last edited on Sun Jan 25th, 2009 09:11 by Lottis |
||||||||||
|
Dr Trevor Marshall Research Team
|
Lottis said "Pregnant women often get infected with fungus" I have several comments about this. Firstly, it is common for physicians to diagnose "fungus" based just on what the inflammation 'looks like'. Second, the acute inflammation of Th1 dysfunction weakens the immune system to the point where an actual fungus may be able to take hold, and even become strong enough to be cultured. The $64,000 question is, of course, whether it is Th1 inflammation or fungus-induced inflammation at the heart of what occurs in pregnant women. Amy just wrote a peer-reviewed paper about Th1 in women, but it is still "in press." meanwhile you might like to look at her Porto transcript at: http://AutoimmunityResearch.org/transcripts/ICA2008_Transcript_AmyProal.pdf |
||||||||||
|
Lottis Health Professional
|
Amy is really doing some groundbreaking work! I am eagerly waiting for the paper to come out. That is going to be very interesting! Back now to the iron. http://www.jimmunol.org/cgi/reprint/178/10/6367 On page 3, at the right column in the journal of immunology, read the section that starts with this sentence; LF levels in PMA-treated PMN supernatants from normal controls and CGD patients were determined by ELISA. There is a dysfunction of the ability to disable the pathogens by requesting their iron, in the CGD patients. |
||||||||||
|
Dr Trevor Marshall Research Team
|
Lottis, You are trying to read too much into these in-vitro experiments, which at best are only a poor simulation of what happens in the diseased body. It is well known that many bacterial strains accrete ferritin. No need to focus on fungus. |
||||||||||
|
Lottis Health Professional
|
Sorry, I am not focus on fungus....that was a side topic.It is the neutrophiles function that caught my interest. I know that pathogens rob us from our iron. That is why I do not want to give iron to my daughter, who has been diagnosed by a hematologist of being low in iron. This doctor suggested iron supplementation, I hesitated, because I knew this. I am trying to find another solution for my daughter and her sick and tiredness. http://curemyth1.org/view_topic.php?id=1072&forum_id=5&highlight=Little+My No doctor wants to even discuss the MP treatment for her. She is also low in her neutrophile counts, so I see a slight connection here. Her B12 is around 400, and I have no clue if B12 supplementation would be good for her. She is in her final year at school, and she is aiming for high grades. That is why I need to understand this subject, you see. -She has to get some help./Lottis |
||||||||||
|
Lottis Health Professional
|
Curcumin in curry, might effect the levels of iron. http://bloodjournal.hematologylibrary.org/cgi/content/abstract/113/2/462 |
||||||||||
|
MarkN Member in Phase 3 
|
When I was researching lactoferrin, yes it keeps iron from the bacteria ... some say it also has a direct antibacterial effect, and I even found some references that it may send a chemical "message" that tells the bugs not to form biofilm. So I don't know what it "really" does, except that it gives me very strong IP, especially where I need it, in the brain. It is also said to "modulate the immune system", which is generally a bad idea, so I don't take it very often. I do feel an anti-inflammatory effect for about 12 hours after taking it. |
||||||||||
|
cschifel Member
|
I have been using the tinted Kabana sunscreen. It is tinted with iron. Should I stop? I use it when I have to go out in the sun or heavy flourescent lighting. I don't now how much iron can be absorbed through the skin. I only put it on my face. Carmen |
||||||||||