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Sallie Q
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Posted: Thu Apr 14th, 2011 01:49 |
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tom wrote: ...... I realize now that Beni and its affect on the VDR can move the protocol ahead. ...................am I not slowing progress down without the more aggressive use of the added ABX?
Tom
it is a common misconception that adding ABX to Benicar = "progress"
If ABX are not added as recommended [i.e. only when symptoms have become negligible for an appropriate period] then addition of ABX is equally likely to slow your recovery by adding stress to an already stressful workload.
being on MP adds meaning to the word 'patient'
Beni and its affect on the VDR is the protocol
the ancient Romans got it right "festina lente"
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tom
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Posted: Fri Apr 15th, 2011 02:48 |
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Hey Sally-Q
Thanks for the info. This seem to be a great confirmation as to info of late. Perhaps I should take further heed to this tip. I have no problem with Beni adding new meaning to the word patient, perhaps I would substitute the word collembola for beni- as this has been my new and most egregious challenge. Still, your confirmation helps with the continued clarity.
Thanks for chiming in.
All the best 
Tom
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tom
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Posted: Sat Jul 14th, 2012 19:11 |
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Cycle: 7 day 7 since reintroduction to ABX
MP Meds: Beni 40mg q6h, Clindy 1/2 (75mg) qod, Mino 1/2 (50mg) qod, Zith 1/2 (75mg) every 10 days.
Non MP Meds: Melatonin 3-4.5 mg as needed
Sun Exposure: -10hrs
The purpose of this thread is connecting the viability of the Marshal protocol in the treatment and eventual resolve of these most egregious skin infections. Although the theory that autoimmune correction caused by the absence of the microbiota would also resolve this anomaly is a sound one. In light of all previous inflammatory problems that I have thus far overcome under the MP the likewise continued application indicates to me that this could as well succumb in like manner.
Nonetheless, based on my limited experience thus far, the viability of this as direct treatment still remains open ended for the following reasons. First, Parasitic infestation of the skin is opportunistic in nature. Subsequently, when the autoimmune system fluctuates as it inevitably will during the course of the healing process so too will the egregious symptoms of collembola (in my case). Such symptoms can overwhelm sleep patterns and risk derailing the entire recovery process altogether. For me this “juggling act” still remains a major challenge to circumvent – far greater than immuno-pathology alone.
Because of this I have also been looking at a preemptive treatment that may minimize this dilemma. Let me say first that I am not indorsing such treatment viability but simply opening such ideas for further discussion by Trevor or others who would like to comment.
The idea is based on a patient who had Morgellons. This patient was not long into the protocol and was undergoing a two stage operation 5- 7 weeks apart. The patient was taking a 1/8 tab of Azithromycin every ten days, and Benicar once every 8 hours as per MP guidelines. The patients 25D was under 12 ngml’s. In an attempt to remain compliant with the outline of the MP during minor surgery the patient requested the substitution of the standard ABX IV to Cleocin. The IV lasted about 45 mins. When the surgery finished the patients Morgellons symptoms had greatly increased. This remained until the patient received a second dose in similar fashion 5-7 weeks later. Shortly after, the patient said that all visual and symptomatic signs of infection disappeared. The patient has not been infected since.
Naturally, after hearing this I became interested in exploring this avenue only to aid in the further navigational process of the MP uninterrupted. I have secured an environment willing to administer a similar blueprint via IV (without the surgery off course).
Although my symptoms of collembola have fluctuated at earlier stages, they have now taken an upward trajectory. Although determined to continue with the MP as a standalone, I remain very guarded in the collembola’s threats to derail the MP process altogether.
Again, let me reiterate I am not challenging the efficacy of the MP (it continues to perform as suggested) I am simply looking for ways to further assist in its full compliance. Equally interesting is how the MP may have assisted with IV in a more targeted approach to morgellons.
Thank you for reading.
Tom
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tom
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Posted: Wed Jul 25th, 2012 21:40 |
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Cycle: 7 day 8 since reintroduction to ABX
MP Meds: Beni 40mg q6h, Clindy 1/2 (75mg) qod, Mino 1/2 (50mg) qod, Zith 1/2 (75mg) every 10 days.
Non MP Meds: Melatonin 3-4.5 mg as needed
Sun Exposure: -10hrs
Although I’ve experienced this collembola infection for around three years this is my first real return to the full abx combo. As expected there have been increased symptoms. Standard IP indicates mild but notable continued improvement with my chronic fatigue infection. However, as suspected, my sensitivity to collembola has increased. Certainly taking some palliative precautions have helped but the trend seems to be upward. In the first 2 weeks I experienced a major resolution in symptoms followed by another week of high activity-followed by weeks of alternating highs and lows. Two weeks ago I broke out in rashes on my face neck and chest-a couple in clear circular patterns. They scratched and burned for quite some time. As a matter of fact it let up just a short time ago.
Figuring that I was nearing the end of the cycle and wanting to maintain my level of IP just the other day, I took my ½ Mino and Clindy then jumped into a very hot sauna. What I didn’t expect was the high level of collembola activity lasting through the night and into the following day.
My theory is although increased IP is the fallout of a healing immune system the temporary stress of this process progressively increases collembola symptoms not decreases them. How long this complication lasts and to what degree of activity, is still an open question. Perhaps there will be a tipping point once full ABX recommendations have been reached and IP slowly begins to diminish. So far I’ve managed to control the collembola symptoms even with this increased IP .
Dr. William Harvey MRF (Morgellons research foundation) with his extensive research concluded the following back in 2007:
“The summary data was extremely revealing, and briefly, showed us consistent abnormalities in immune function, chronic systemic inflammation mult-system involvement…” Hmmmm
He theorized that the culprit was some “parasitic worm” which had invaded the immune cells. He passed away from a heart attack a couple of months ago perhaps never reviewing the studies here on CWD bacteria
Continued progress for discussion will be posted
TomLast edited on Wed Jul 25th, 2012 21:43 by tom
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Cynthia Schnitz
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Posted: Tue Jul 31st, 2012 15:46 |
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tom wrote: In the first 2 weeks I experienced a major resolution in symptoms followed by another week of high activity-followed by weeks of alternating highs and lows.
I am not sure what this first 2 weeks refers to. After what? I assume it must have taken you some time to get to your current ABx level. Cynthia
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tom
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Posted: Wed Aug 8th, 2012 23:46 |
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Cycle 9 day 9 since the reintroduction of ABX
MP Meds: Beni 40 mg q6h, Clindy ½ (75mg) qod, Mino ½ (50mg) qod, Zith ½ (75mg) every 10 days
Non MP Meds: Melatonin 3-4.5 as needed
Sun Exposure: 10hrs -
Hi Cynthia,
Thanks for bringing to light the obvious oversight. So let me clarify. I restarted the protocol with the introduction of ABX on May 8th. I started with Z and moved to clindy and Mino as IP allowed. Initially at the point of increase there was a detectable level of increased collembola. This stayed pretty much constant until I reached or came close to approaching the ½ dose mark across the board (about July 5th). At this time my skin symptoms decreased notably and lasted for several weeks thereafter. On or about the last week in July I began experiencing increased symptoms once again but this time far more consistent and of a much higher level than I had experienced since the summer began-this continues at present.
Here are some conclusions that I have drawn thus far:
First, Increased Immunopathology will stimulate collembola as it responds to a weakened immune system during the healing process. This maybe further supported by the fact that the highest collembola activity is localized around areas of highest IP (head neck and chest), correlated around Nuro and chest IP due to lingering brain fog and valley fever (infection of the lungs) acquired while back in Phoenix.
Although the skin symptoms are my main challenge in the recovery process I remain somewhat uncertain as to its trajectory. Could I assume that as long as I experience IP then increased collembola will remain a very challenging byproduct? Perhaps this will be directly proportionate to the level of IP that I’m experiencing. Staying compliant with the MP during such symptoms becomes ever more challenging when compounded with standard IP. This in itself may not become a deal breaker, and well worth the sacrifice if the trajectory remains shortened. Seeing myself as a test pilot of sorts I’ll continue to plod along…for now.
Thanks for reading
Tom
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Dr Trevor Marshall
Foundation Staff
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Posted: Thu Aug 9th, 2012 00:06 |
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Tom,
Have you read the MP guidance recently?
http://mpkb.org/home/mp#starting_a_patient_on_the_marshall_protocol
We actively discourage the use of Zith, and the use of antibiotics unless you need them for palliation or additional IP.
Things have changed a lot over the last few years, as the science fully firmed up.
..Trevor..
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tom
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Posted: Thu Aug 30th, 2012 20:17 |
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MP Meds: Beni 40 mg q6h (Since Aug 2nd)
None MP Meds: Melatonin 3mg as needed
Sun Exposure: 10+ Hrs
V-D 12.9 ng/ml
Thanks Trevor for the link. I plan to reproach with more caution.
I dropped off the ABX (August 2nd) due to a resurgence of my Valley Fever (Fungal infection of the lungs). It was my fault as I pushed too hard on the MP. I’ve been on the beni since with diminished symptoms of my collembola. Once again skin activity diminishes when IP diminishes. I plan to return with the more modified version of the MP as posted above in the next few days. Because of its sulfur properties bactrum will be introduced for starters.
I still would like to do an exploratory probe with the treatment mentioned (see July 14 post above). I had it all scheduled with my doctor but she got cold feet and aborted the treatment. I’ll try to reattempt with a new lab in the weeks to come. Naturally, I will post all the details as it unfolds.
Thank you for reading
Tom
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Dr Trevor Marshall
Foundation Staff
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Posted: Thu Aug 30th, 2012 23:49 |
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New article says that Rosacea is due to a bacteria inside a worm-like-mite which lives in everybody's skin. I don't know if I like to contemplate that possibility, its pretty creepy, but it could well be accurate:
http://www.sciencedaily.com/releases/2012/08/120829195121.htm
If they can find these worm-like-mites in healthy skin, then why can't they find these parasites in Morgellons skin?
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k
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Posted: Fri Aug 31st, 2012 08:39 |
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Lol! I heard about this article and its contents on the radio on this morning when I woke up. And what I thought was "Pfft! If I can cope with the concept of the human microbiome / 90% of cells in my body being non-human, then worm-like-mites living in my skin is nothing!".
(It did take me a while to get used to the concept of the human microbiome!, which I initially found a bit creepy)
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tom
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Posted: Sat Oct 6th, 2012 20:00 |
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Mp Meds: Beni 40 mg Bactrim 1/4 tab (37.5)qod 3 weeks
Non MP Meds: Melatonin 3mg as needed.
Sun Exposure: 10-15 hrs
VD 12.9 ng/ml
This is a fascinating article. Like these tiny Demodex mites there are mites that can actually travel on the backs of other mites. Subsequently one would theorize that you would have the involvement of more complex or serious infections perhaps due to a combination of particular bacteria or fungus's. As in my situation collembola was a new parasite that infected my body shortly after contact in a highly infectious situation. Once penetration of the skin was made, manifested by lesions and bites developing on my body, symptoms progressed even when relocating. I would assume that the bacteria spread attracting more skin parasites continuing the cycle. I immediately began to contend with all sorts of physiological and well as neurological symptoms. Temperature regulation disorders, anxiety attacks, fear and depression. At first I thought this was simply caused as a standalone to the stress I was experiencing. But this carried on those day's when symptoms were light. I could only conclude “unscientifically” that this maybe attributed to a heavy release of endotoxins into the system. There are many similarities between Collembola and Morgellons and the Demodex mites or collembola are not alone. Scientist figures their maybe tens of thousands of mites most that have yet to be identified. Subsequently could this mean more than one bacteria, fungus or parasite?
What made the Rosacea a particularly interesting article is that some of my symptoms during heavy IP have recently taken on similar characteristics around the neck and face (large areas of painful and burning splotching. This immediately begins to fade when the ABX half life is exceeded. This is in addition to other Collembola infected areas of the body which manifest different but correlated symptoms. Although the oral treatment has been Bactrim DS which I’m taking now I surprisingly enough found similar responses to Minocycline- all in conjunction with the application of the MP.
What is particularly interesting is that no positive fallout to IP was seen until now-precisely when my valley fever symptoms began to abate. Is it possible that the immune response has just now begun to prioritize my skin issues? I’m curious to see how this plays out in the weeks to come.
Thanks for reading this most grizzly article.
TomLast edited on Sat Oct 6th, 2012 20:04 by tom
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